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I am taking Gleevec after surgery, to remove 15ccmm tumor. It has been 7 months and I am tumor free. The main side effects of taking Gleevec has been abdominal bloating with pain, at times severe. I am able to handle the side effects for the most part, but when it becomes too much, I just reduce my dose for a few days until I feel better, normally or for two days a week. I was on Gleevec for 5 months prior to surgery. It didn't reduce the tumor, but it stop its growth and reduced the density considerably.



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I am 46 years of age and my doctor put me on Gleevec to remove the tumor I have. Actually I had a surgery last year and I am taking Gleevec now to prevent possible reoccurrence of the tumor. I haven't had any side effects except for minor abdominal pain. But it is not significant in comparison to the benefits the medication provides. I am on Gleevec for more than 9 months and there is no single sign of the tumor to reappear. It goes without saying that my diagnosis was a shock for me. I thought that my life was over and now I am just had to for my death to come. But it was 6 years ago and at that time I knew nothing about cancer. Now I know a lot. I know that life goes on and the main treatment is in our minds. If we strongly decide to live and to find a solution, we'll do that. Even in most difficult situations hope works out miracles! It was my friend who told me about Gleevec. She is the sunniest person I know and you will never think that this charming woman has cancer if you meet her on the street. She is full of life and strong belief. And it is she whom I owe a great part of my success as I borrowed hope and belief from her. She was also taking Gleevec, but prior to the surgery and it stopped her tumor from growth. Now she's also on it to consolidate the success of the operation and I pray for her. I pray for everyone with same problems.

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CML-CP has been very significantly prolonged by imatinib therapy.

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Preoperative diagnosis of malignancy is unusual, accounting for about 30% of cases.

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Combination treatment of imatinib with mTOR inhibitors could be effective in cases where Bcr-Abl mutants do not cause complete resistance to imatinib ().

Patients who received adjuvant TKIs had a higher disease stage and were more likely to receive adjuvant chemotherapy before starting adjuvant TKIs.

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Several types of mutations in the Abl gene can be found in chronic myeloid leukemia patients resistant to STI571, and they can pre-exist to the onset of treatment.

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LC3B proteins were detected by western blot and Hsp60 was used as loading control ( b).

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Those speculations were confirmed with a two-fold increase in inhibitory action against Bcr-Abl T315I mutated kinase and the silver lining was the of the substance (named '19a') appeared to have decreased, allowing for smaller doses with the same potency.

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Low antithrombin III concentrations, thrombocytosis, decreased platelet survival, and spontaneous platelet aggregation causing coagulation abnormalities have also been reported.

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This drug should be strictly taken at regular intervals.

Evaluating the Impact of a Switch to Nilotinib on Imatinib-Related Chronic Low-Grade Adverse Events in Patients With CML-CP: The ENRICH Study.

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CASE OF GASTROINTESTINAL STROMAL TUMOR OF THE STOMACH WITH MULTIPLE LIVER METASTASES SURVIVING LONG-TERM WITH MULTIMODAL TREATMENT.

Imatinib may also be used to treat other types of cancer, such as with the presence of the Philadelphia chromosome.

You should not become pregnant while taking SUTENT.

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FCERI mediated MAPK activation, organism-specific biosystem Formation of the LAT signaling complex leads to activation of MAPK and production of cytokines.

Sequencing of the region encoding the aa that bind ATP and imatinib revealed acquired mutations which lead to substitutions of amino acids which are important for specific binding of imatinib.

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Imatinib is generally incorporated into the treatment of patients with Ph1-positive ALL because of the responses observed in monotherapy trials.

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Patients with B-precursor ALL and high WBC counts at diagnosis have an increased risk of treatment failure compared with patients with low initial WBC counts.

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Development of fluid retention as an adverse effect of imatinib in a 64-year-old woman with metastatic small bowel GIST.

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Which patients with CML should proceed directly to allogeneic stem-cell transplantation, a potentially curative treatment, without first undergoing a trial of imatinib?

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Bone marrow suppression (myelosuppression) including a decrease in the number and effectiveness of platelets (thrombocytopenia), red blood cells (anemia) and white blood cells (neutropenia).

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The TKI, which inhibits activity of BCR-ABL1 kinase, was the first of its kind to be successfully used in the treatment of CML.

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Therapy is based on the extent of disease and prognosis for limited disease is generally very good.

In addition, one study showed an improvement in survival after second HSCT if acute GVHD occurred, especially if it had not occurred after the first transplant.

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Shorter halving time of BCR-ABL1 transcripts is a novel predictor for achievement of molecular responses in newly diagnosed chronic-phase chronic myeloid leukemia treated with dasatinib: Results of the D-first study of Kanto CML study group.

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After 3 months of imatinib therapy the MRSS was 29.

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